AMATEKA

Menya amavu n’amavuko y’inzoga ya Mützig

November 30, 2023
professormaurice
539 Comments
Abarebye: 18,204

HAKIZIMANA Maurice

Kimwe na Primus-Gahuzamiryango twavuzeho mu nkuru iheruka(kanda hano),inzoga ya Mützig (benshi basoma Mitsingi)ni kimwe mu byengwa n’uruganda BRALIRWA. Hari bamwe mu bashima iyi nzoga bavuga ko ifite uburyohe hakaba n’abayitinya bemeza ko ngo yaba ijanjagura umutwe ku wayinyoye. Mbese ubundi iyi nzoga ikomoka hehe?

Amateka ya “Mitsingi” mu ncamake

Inzoga ya Mützig yatangiye kwengwa mu mugi witwa Mützig mu ntara ya Alsace mu Bufaransa hafi y’umupaka uhuza iki gihugu n’u Budage. Urwengero rwa Mützig(Brasserie Mutzig) rwatangiye gukora mu mwaka wa 1810 rushinzwe na Antoine Wagner.

Uru rwengero rwaje kwiyunga n’izindi eshatu zo mu ntara ya Alsace zose zihinduka ikompanyi imwe yitwaga ALBRA (Alsacienne de Brasserie).

Mu mwaka wa 1972 ALBRA yaguzwe na sosiyeti mpuzamahanga HEINEKEN ikomeza kwenga inzoga ya Mützig. Abatangije uruganda rw’iyi nzoga bakomeje kuyenga ndetse no muri iki gihe Mützig iracyakorwa mu Bufaransa mu ruganda rwitwa Fischer rw’ahitwa Schiltigheim.

Mützig mu Rwanda

Uruganda rwa BRALIRWA (Brasserie et Limonaderie du Rwanda) rwatangiye kwenga Mützig mu mwaka wa 1987. Batangiye bakora inzoga yo mu icupa rya santilitiro 65. Nyuma y’imyaka itatu haje na Mützig ntoya zo mu macupa ya santilitiro 33. Mu mwaka wa 2001 habonetse Mützig à pression cyangwa draught ya yindi basuka mu birahure binini ivuye mu ngunguru. Usibye mu Rwanda ahandi muri Afurika bakora inzoga ya Mützig ni muri Repubulika iharanira Demokarasi ya Kongo, Kameruni na Kongo Brazaville.

Mu myaka mike ishize uruganda BRALIRWA rwagerageje ubwoko bushya bwa Mutzig Light ndetse na Mutzig yo mu icupa rya santilitiro 50 bita Babu G ariko mu by’ukuri ayo moko uko ari abiri ntabwo yakiriwe neza n’abakunzi b’agasembuye.

Niba waranyoye kuri iyi nzoga mbere y’imyaka 30 ishize,tubwire icyo uyibukiraho?Itandukaniye he n’iy’ubu?

Iyi si,

NB: Inyandiko dukesha mukerarugendo.rw

Mugire ibihe byiza,

IYI SI,
Komeza gukurikira uru rubuga rwigisha uzamenya byinshi ukeneye mu buzima bwa buri munsi.
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539 thoughts on “Menya amavu n’amavuko y’inzoga ya Mützig”

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  505. After the 5 min warm-up interval, subjects continued to warm-up for an additional 20 s adopted by a 6 s
    acceleration part, throughout which they pedaled as fast as attainable
    towards no resistance to realize peak cadence. Instantly at the end of this phase,
    a load equal to 7.5% of body weight was utilized to the flywheel
    and subjects pedaled as quick as attainable for 30 s. Knowledge was recorded and
    saved utilizing the Velotron Wingate software program (Racer-Mate, Seattle, WA).

    The EPI group consumed one capsule containing one hundred mg of 98% pure (–)-epicatechin twice daily (200 mg total).
    Members had been instructed to eat one 100 mg capsule within the morning and one a hundred mg capsule in the afternoon or evening.

    Myostatin, also identified as development differentiation factor
    eight (GDF8), is a transforming growth factor-β (TGF-β) family member that potently inhibits skeletal
    muscle improvement [1]. The biological perform of myostatin turned
    evident when mice homozygous for a deletion of myostatin gene exhibited a dramatic improve in skeletal muscle mass,
    with individual muscle groups enlarging to approximately twice their normal size [1].
    In addition to its effect on skeletal muscle, myostatin has been demonstrated to play
    a major position in the regulation of bone metabolism by suppressing bone formation [6]
    and stimulating bone resorption [7]. Indeed,
    myostatin null mice characterize not only a doubling of muscle mass,
    but additionally enhanced bone mineral density (BMD) and bone regeneration [6,8].
    Duchenne muscular dystrophy (DMD) is an incurable illness that causes
    the lack of muscle tissue. Myostatin inhibition has been proven to increase muscle mass in canines and
    mice with DMD, suggesting that the potential of myostatin inhibitors should be investigated in human sufferers with DMD [7, 16].
    In the present study, the effect of FOR supplementation have been compared with a macronutrient-
    and energy-matched placebo on indices of muscle dimension and energy throughout two weeks of single-leg immobilization in young,
    healthy males.
    The serum from the remaining separation tube was separated,
    labeled, and stored in micro centrifuge tubes at -80°C for later
    evaluation. Following completion of the study, samples were analyzed
    for active testosterone, free testosterone, and cortisol with an enzyme immunoassay (EIA)
    assays using Goat-Anti-rabbit IgG (GARG) coated microplates.
    Assays were carried out utilizing a Jitterbug microplate shaker (Boekel Scientific-Philadelphia, PA) and a Tricontinent Multiwash Benefit microplate washer (Grass
    Valley, CA).
    A myostatin inhibitor prevents myostatin from binding to its receptor, thereby repressing the downstream Smad-pathway [11].
    By supplementing it, you could expertise benefits
    in muscle growth and strength and improvements in vascularity, blood flow, and endurance.

    Training with heavy weight likely offers a feedback stimulus that inhibits myostatin,
    partially to permit for practical muscle development processes to
    kick in. If this didn’t occur, we would go back to the health club time and
    again with minimal gains to point out for it. Coupled with the myostatin inhibition that resistance coaching itself elicits,
    there may be no room for one more inhibitor
    to work. The study concludes that additional exams
    are wanted to see whether or not creatine, leucine and HMB are synergistic with respect to myostatin inhibition (and other progress pathways) or not.

    Carlon Colker, M.D., found myostatin inhibitors in egg yolk
    from fertilized chicken eggs. Dr. Colker developed a process to optimize the myostatin-inhibiting exercise in yolk membranes,
    and the ensuing product is MYO-T12. It Is one of the talked-about
    myostatin inhibitors because of its potential to scale back myostatin ranges,
    thereby permitting for elevated muscle progress and improved energy.

    Epicatechin also promotes nitric oxide manufacturing, which improves blood
    flow and nutrient supply to muscles, enhancing overall muscle
    operate and endurance. Another potential concern is that elevated muscle growth will
    result in an elevated threat of harm due to increased stress on the muscle fibers.

    Myostatin is produced by the muscle tissue of the heart,
    and harm to the heart causes it to be released into the
    bloodstream. Once within the bloodstream, myostatin reaches
    the muscle tissue and causes them to weaken over time.
    As a result, patients with coronary heart illness often additionally experience signs of muscle atrophy (muscle loss) [10].
    Bodily train, significantly weightlifting, can naturally decrease
    myostatin activity. Research show that resistance coaching improves muscle fibers,
    reduces body fat, and stimulates skeletal muscle hypertrophy.
    Myostatin inhibitors block or decrease myostatin activity and “unlock” your muscle-building potential.
    By suppressing myostatin expression or its signaling pathways,
    these inhibitors enable your physique to create extra muscle fibers and reduce muscle
    atrophy.
    Myostatin is considered an inhibitor of satellite cell activation and as a
    result skeletal muscle hypertrophy. One promising
    complement which has suppressed blood levels of myostatin by
    44% is a proprietary bioactive ingredient, Myo-T12, which is follistatin derived from fertile hen egg yolk isolate.
    Therefore the aim of this research was to analyze the consequences of MyoT12 on skeletal muscle progress and power in recreationally skilled individuals.
    Over 20% of most cancers deaths in human patients is attributable
    to cachexia, a symptom of most cancers that causes the lack of muscle
    and fat despite enough vitamin.
    The samples were then centrifuged at 3000 rpm for
    10 min at four °C to collect the supernatant.

    The levels of the neurotransmitters acetylcholine (ACh), glutamate (Glu), gamma-aminobutyric acid (GABA), and serotonin (5-HT), that are related to fatigue, were measured using ELISA following the manufacturer’s directions.
    The absorbance at 550 nm and 340 nm was measured utilizing a spectrophotometer or microplate reader.
    The kits used on this research were evaluated for consistency of performance throughout totally different batches of kits by
    restoration tests with recoveries starting from 90%-110% and calibrated
    by standards. The tools used had a measurement accuracy of 0.01% and was
    calibrated before each experiment. Ltd. (Wuhan,
    China), and cultured in DMEM supplemented with 10% FBS and 1%
    penicillin/streptomycin (Cell-specific DMEM; Procell, Wuhan, China) in an incubator at
    37 °C with 5% CO2.
    For instance, injection of ACVR2B-Fc fusion protein to wild-type mice induced a dramatic enhance (40%-60%) in muscle mass in just 2 weeks
    [37], an effect larger than that obtained after treatment with D76A myostatin propeptide or monoclonal
    anti-myostatin antibody. Similarly, administration of
    ACVR2B-Fc fusion protein to a mouse mannequin of osteogenesis imperfecta (OI) brought on important
    increases in each bone and muscle mass that
    had been close to those seen in wild-type mice [38]. Importantly,
    the soluble ACVR2B receptor exhibited anabolic effects even in myostatin null mice,
    indicating the existence of different TGF-β members of the family that
    perform to restrict muscle growth [37]. After disuse
    atrophy was induced, the decreased muscle CSA and LM of the immobilized leg weren’t
    recovered within 2 weeks of returning to regular activity in each
    groups (Fig 2). Similar to the period of
    immobilization, we could not observe any impact of FOR ingestion on skeletal muscle through the restoration section. A earlier examine reported that 2 weeks of immobilization was adequate to induce anabolic resistance–decreased response of MPS to anabolic stimuli–in younger adults [16].
    Thus, there’s a risk that the impression of FOR was not effective sufficient to recover
    the lack of muscle dimension with anabolic resistance induced
    by immobilization or needed an prolonged recovery interval.

    Follistatin (FST) and its related FST-type molecules are naturally antagonists to several TGF-β
    proteins, and are extensively identified inhibitors of
    MSTN [70, 71]. FST binds to mature MSTN with high affinity and inhibits its binding to ActRIIB,
    but does not work together with proMSTN [20, 72].
    The N-terminal α-helical domain of FST interacts immediately with a kind I receptor binding web
    site of MSTN, inflicting inactivation [73]. Consequently,
    FST missing its C-terminal peptide or fragments of the N-terminal area show comparable
    inhibitory results [73–75].
    However, but extra research signifies that myostatin may merely be an anti-hypertrophy agent quite than the
    main muscle atrophy trigger. The totally different methods present there
    are two particular periods of muscle growth development that myostatin regulates
    closely. Obviously, anyone trying to construct muscle
    would be excited about a safe method to inhibit
    the myostatin protein, even just a little, to permit for larger muscle development gains.
    Some “supplements” include follistatin, an experimental compound being studied for
    blocking the actions of myostatin. Potential
    unwanted effects include muscle imbalances, elevated injury danger, and unknown long-term effects
    on body composition and bone metabolism. Seek The Advice Of
    a healthcare skilled before use, especially when you have
    underlying health conditions. There ARE myostatin dietary supplements on the market right now that work
    at lowering your body’s capacity to provide
    this protein.
    Whereas myostatin inhibitors like peptides and SARMs are gaining recognition, you can even lower your myostatin ranges
    naturally through meals and supplements. And whereas the proof is
    both anecdotal or comes from animal models, these natural choices
    are nonetheless a fantastic place to begin out.
    Myostatin, also recognized as growth differentiation factor 8 (GDF-8),
    is a member of the TGF-beta superfamily. Whereas that sounds like
    the other of what you’re on the lookout for in your fitness story,
    myostatin actually performs a vital role in maintaining muscle homeostasis
    and preventing uncontrolled growth.
    SM produces several bioactive proteins, including cytokines, and quite a few other
    peptides collectively referred to as “myokines”. Skeletal myofibers
    produce a plethora of myokines, which exert auto-, para,
    and/or endocrine results. Since myokine secretion is mostly regulated by train,
    it has numerous advantageous results on metabolic, cardiovascular, and mental
    health (Manole et al., 2018).
    Nonetheless, Smad2 expression was not considerably
    decreased by the peptide remedy (Supplementary Determine S7).
    These findings indicate that Ac-MIF1 or Ac-MIF2-NH2
    peptides promote myogenesis by rising the expression of myogenic marker genes.
    Regardless Of the suppression of MSTN mRNA expression, we didn’t observe any changes in amino acid-stimulated myotube
    protein synthesis, as indicated by the incorporation of the tyrosyl-tRNA analogue Puromycin into newly synthesised peptides.
    Subsequently, longer duration of treatment with UA may be required to research the results of sustained myostatin suppression on protein synthesis.

    Supporting this speculation, it has previously been demonstrated that persistent UA administration upregulates the exercise
    of mitochondrial respiratory complexes I and II within the skeletal muscle of muscular dystrophic or
    HFD-fed mice [17, 19]. In the postprandial state,
    as much as 50% of the glucose that is taken up by skeletal muscle undergoes oxidation, demonstrating the importance of this mechanism
    in glucose homeostasis [37].
    We hypothesized that FOR supplement would attenuate the decline in muscle measurement during immobilization, which would coincide with
    a reduction of circulating myostatin and alterations of molecular markers favoring
    a less catabolic state. Anaerobic power and anaerobic capability elevated from training with no distinction between the teams.
    Moreover, although there was an increase within the fatigue index over time, no
    group differences were noticed. The change in peak anaerobic power noticed in this
    examine is analogous in magnitude to a previous study (33).
    Vera-Ibañez et al. (33) studied the impact
    of high-intensity biking for 4 weeks on peak energy output during
    the Wingate take a look at. They observed an increase in peak energy output of ~15% whereas the present study noticed an increase of ~11%.

    Transgenic overexpression of FST leads to a 2- to 3-fold increase in muscle mass by way of hypertrophy
    and hyperplasia [21]. However, the rise in muscle mass just isn’t solely attributed to
    blocking MSTN, as FST additionally inhibits the activities of a quantity of TGF-β relations, some of them play a
    job in limiting muscle mass [76]. FST-overexpressing MSTN-null mice show an even more extreme
    fourfold increase in muscle [77]. Contrarily, FST-null mice
    have lowered muscle mass at birth and perish inside a number
    of hours [78]. Delivery of FST-coding mRNA gene remedy and AAV, in addition to
    follistatin peptide derivatives, have all been shown to provide substantial muscle improve in animal fashions [35, 79, 80].
    Scholar Rock has submitted its anti-myostatin antibody apitegromab for FDA approval, just months after reporting that
    it hit its primary endpoint in section III for spinal muscular atrophy (SMA).

    Approval would upend twenty years of failure for myostatin pathway inhibitors,
    a category of medication that grows skeletal muscle.

    As time evolves, clinical research will hopefully
    verify the putative advantages of epicatechin for energetic
    people. Curiously, studies that discovered a significant interplay between the lively treatment and myostatin levels have been investigating
    epicatechin-enriched green tea extracts.
    Such findings indicate that green tea catechins work synergistically to reinforce
    skeletal muscle adaptation. In conclusion, the article also highlighted
    studies on numerous other supplements, together with brown seaweed,
    spirulina, cocoa, epicatechin, and Vitamin D. Nevertheless, the
    findings were largely inconclusive.
    Two molecules are epimers of one another once they have no less than two asymmetric carbon atoms but differ by configuration at only a sort of carbons.
    Basically, epimers are equivalent molecules with the identical asymmetric carbon atom (and its bonds) rotated in another way
    in house (see image beneath for reference). To get essentially the most out of
    the supplement, Enhanced Athlete recommends taking 1-2 capsules per day with meals.
    The pure muscle constructing supplement is free from synthetic fillers, dyes,
    chemicals, and deceptive proprietary blends, and is made within the Usa of America.

    Apparently, MSTN protein ranges had been considerably decrease in Ac-MIF2-NH2-injected muscular
    tissues (Figure 6C), and ACVRIIB protein ranges were lower in Ac-MIF2-NH2-injected
    muscle tissue (Supplementary Figure S8). In addition,
    muscle fiber widths had been significantly higher in Ac-MIF1-treated muscles than only CTX-injected muscular tissues (Figure
    6D). Taken collectively, Ac-MIF1 and Ac-MIF2-NH2 stimulate muscle regeneration in injured muscle
    tissue.
    One Other talked-about technique of inhibiting myostatin is through the consumption of
    epicatechin, a member of the flavonoid household of chemicals that management pigmentation in plants [31].
    In each people and animals, myostatin is a hormone
    that acts as a kind of “brake” that tells muscle tissue to stop rising, which helps to
    forestall them from getting too massive. Uneven muscle development is
    a possibility when you don’t persist with a regimented coaching and recovery schedule.
    Unchecked muscle development can exhaust tendons and ligaments, increasing
    the danger of injury. SwissChems is a trusted name in the peptide world,
    and their Myostatin delivers a potent formulation designed to target myostatin inhibition instantly.
    However, Welle did mention some unwanted facet effects within the
    mice come the top of the study.
    Nevertheless, SOD1 null mice, a mannequin of amyotrophic lateral
    sclerosis, didn’t exhibit any improvements in survival
    (despite enhancements in muscle mass) when uncovered to myostatin inhibitors [56].
    In contrast to the above stories, therapy of the myotubularin-deficient mouse, a mannequin of X-linked myotubular myopathy, with ActRIIB-Fc did lead to
    transient increases in muscle mass and energy and a 17% improve
    in survival [60▪▪]. Angiotensin II serves as an endogenous
    protein in muscle tissue, which inspires skeletal
    muscle loss. Angiotensin II depolarizes mitochondrial membranes, resulting in elevated mitochondrial
    ROS era and inducing oxidative stress in muscle tissue [95].

    Hence how some myostatin inhibor supplements are receiving poor critiques on-line with prospects complaining that they didn’t discover a factor.
    Now, to turn out to be top-of-the-line bodybuilders
    on the planet like Flex, you’ve received to have past distinctive genetics, use anabolic steroids and practice onerous within the gym for a really long time.
    In my opinion, the gathering of blood samples solely on days 14 and 28 must be clearly acknowledged as a
    limitation of the manuscript. Standardize the writing of the names of examine phases throughout the
    manuscript – sometimes capitals are used as a primary letter of the name of the phases, and in the different
    parts of the manuscript lowercase is used.
    The number of myostatin-positive SC–myostatin inhibits the progress of SC into the
    S phase of the cell cycle [9]–increased after 2 weeks of immobilization in each groups.
    More studies are needed to discover out the relationship between disuse-atrophy and myostatin-positive SC.
    Nevertheless, it was in line with the earlier research reporting the blunted response of the decline in the number of myostatin-positive SC to resistance
    exercise in older adults, who could have an impaired myogenic
    capacity compared to young [34].
    Nonetheless, it is important to observe that using myostatin inhibitors in enhancing athletic performance is considered unethical and should have
    adverse well being results. Myostatin (or progress differentiation factor-8, GDF-8)
    is a negative regulator of muscle progress and a member of the remodeling
    progress factor-β family [7]. Varied molecular mechanisms regulating
    protein synthesis and degradation are influenced by myostatin [8].
    Myostatin additionally negatively regulates the activation and self-renewal of satellite
    tv for pc cells (SC) by inhibiting the progress of SC into the S section of the cell cycle [9].

    In addition, myostatin reduces IGF-Akt pathway exercise resulting in decreased protein synthesis initiation and elevated translocation of Forkhead box protein O1 (Foxo1) into the
    nucleus. Earlier research reported an elevated myostatin mRNA expression in skeletal muscle after 5
    days of single-leg immobilization [10] and
    an increase in myostatin mRNA and protein expression after 3
    days of unilateral decrease limb suspension in wholesome younger males [11].
    Whereas Jones et al. [12] showed no change in myostatin mRNA expression after 2-wk of single-leg immobilization,
    though there was an elevated tendency of myostatin mRNA.

    Statistical analyses have been carried out by utilizing a separate 2 × 2 (group × time point) two-way mixed mannequin analyses of variance (ANOVA) for
    each criterion variable. If a gaggle by time point interaction existed, a
    separate one-way ANOVA for every group and time
    point was performed to discover out simple primary results.
    All statistical procedures were performed using the SPSS
    Statistics 22.0 software program (IBM, Armonk, NY). Members warmed-up for 5 min at
    a work price equal to 75 Watts on the Velotron cycle ergometer (Racer-Mate,
    Seattle, WA).

    References:

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